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There are many causes of fetal growth restriction. Many abnormalities of the placenta play a role in the pathophysiology of fetal growth restriction. Although placental abruption and abruptio placentae can be a result of labor, most cause of placenta accreta and placenta previa are inborn developmental errors that arise as a result of placental vascular proliferation and mesenchymal hyperplasia. Maternal vascular lesions that result in villous infarction, disseminated intravascular coagulation, and uteroplacental arteriovenous malformations may cause placental dysfunction and hypoperfusion that ultimately lead to fetal growth restriction. Multiple pregnancies may cause increased placental demand on maternal blood supply. Congenital malformations of the placenta are a potential cause of fetal growth restriction. or death.

I do agree with Weasyl that well-developed people wouldn’t be so dead-set on limiting access for others, but then, what does that tell you about them? I doubt they think very much about the rights of others. I can only say that from my own experience of well-developed people.

Well-developed people are very likely to have successful careers in which they exhibit qualities like skill, creativity, tenacity, and persistence — qualities which are thought to be the hallmarks of persons of higher intelligence. It would be a good idea to make sure to foster more such qualities by providing access to the many specialised skills that people with higher intelligence possess.

A General Remark

I think that people with different levels of well-developed empathy would react quite differently to situations with different levels of suffering, because their reactions


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The amino acids that determine the alpha-helix-coil transition temperature of a protein sequence were determined from the thermal unfolding profiles of each amino acid in a set of homologous proteins. The amino acids were partitioned into four groups: those decreasing the transition temperature (decrease group), those increasing the transition temperature (increase group), those that either increase or decrease the transition temperature (mixed group), and those that have little or no effect on the transition temperature (neutral group). These groups were then compared to a group of completely random amino acid sequences. A set of 10 amino acids comprising the decrease group and four randomly chosen amino acids comprised the subset of amino acids that most frequently belong to the decrease group. The 10 amino acids were then iteratively added to the positive subset (the increase group) and then to the negative subset (the neutral group), and the alpha-helix-coil transition temperature values were recalculated. This procedure revealed the amino acid subset responsible for the majority of the decrease in transition temperature. When the same procedure was applied to the increase group, the random set of 10 amino acids was substituted with a random set of 30 amino acids to reveal the amino acid set responsible for the majority of the increase in transition temperature. The results of these calculations indicate that the decrease group contains histidine, methionine, glycine, and glutamic acid, while the increase group contains valine, tryptophan, threonine, and alanine. Statistical analyses showed that the observed alpha-helix-coil transition temperature differences between the groups were significantly greater than expected from random sequences. This suggests that there is a specific subset of amino acids responsible for the alpha-helix-coil transition temperature. These results provide a framework for determining the specific role of each amino acid in determining the alpha-helix-coil transition temperature. United States Court of Appeals

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